Bhupesh Prusty

Bhupesh K. Prusty, PhD, is the Group Leader in the Department of Microbiology, Julius Maximilian University of Wuerzburg, Germany. His research expertise includes the molecular mechanisms of latency and activation of Human herpesvirus 6 and the finding of the chromosomal integration and inheritance of Human herpesvirus 7.

Education

Dr. Prusty earned his doctorate in Biosciences from Jamia Millia Islamia, New Delhi, India.

Awards

• 2015 - Koichi Yamanishi Young Investigator Award for Excellence in Basic Science given by the Human herpesvirus 6 Foundation
• 2016 - Ramsay Award Program grant recipient, sponsored by the Solve ME/CFS Initiative for his work on Human herpesvirus 6 mediated mitochondrial modulation and its association to ME/CFS.

EUROMENE

Dr. Prusty is a substitute member of the Biomarkers Working Group of ME/CFS across Europe on behalf of EUROMENE, a European Union COST Action CA15111 not-for-profit research organization committed to tackling the cause and treatment for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Notable studies

• 2018, Chronic viral infections in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) (Full text)
• 2020, Human Herpesvirus-6 Reactivation, Mitochondrial Fragmentation, and the Coordination of Antiviral and Metabolic Phenotypes in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (Full text)

Talks and interviews

• SMCI 2016 Ramsay Team 5 Research Update (Interview with Solve ME/CFS Initiative)

Online presence

• PubMed
• ResearchGate
• Loop
• Twitter
• LinkedIn

See also

• EUROMENE
• Solve ME/CFS Initiative
• Human herpesvirus 6 Human Herpesvirus 6

Learn more

• Dr Bhupesh Prusty - Solve ME/CFS Initiative
• HHV-6 Mediated Mitochondrial Modulation and Its Association to ME/CFS - Project summary for Solve ME/CFS Initiative
• Bhupesh Prusty earns Young Investigator Award for Excellence in Basic Science
• Jamia Millia Islamia

mitochondria Important parts of the biological cell, with each mitochondrion encased within a mitochondrial membrane. Mitochondria are best known for their role in energy production, earning them the nickname "the powerhouse of the cell". Mitochondria also participate in the detection of threats and the response to these threats. One of the responses to threats orchestrated by mitochondria is apoptosis, a cell suicide program used by cells when the threat can not be eliminated.

See myalgic encephalomyelitis.

See chronic fatigue syndrome.

mitochondria Important parts of the biological cell, with each mitochondrion encased within a mitochondrial membrane. Mitochondria are best known for their role in energy production, earning them the nickname "the powerhouse of the cell". Mitochondria also participate in the detection of threats and the response to these threats. One of the responses to threats orchestrated by mitochondria is apoptosis, a cell suicide program used by cells when the threat can not be eliminated.

myalgic encephalomyelitis (M.E.) - A disease often marked by neurological symptoms, but fatigue is sometimes a symptom as well. Some diagnostic criteria distinguish it from chronic fatigue syndrome, while other diagnostic criteria consider it to be a synonym for chronic fatigue syndrome. A defining characteristic of ME is post-exertional malaise (PEM), or post-exertional neuroimmune exhaustion (PENE), which is a notable exacerbation of symptoms brought on by small exertions. PEM can last for days or weeks. Symptoms can include cognitive impairments, muscle pain (myalgia), trouble remaining upright (orthostatic intolerance), sleep abnormalities, and gastro-intestinal impairments, among others. An estimated 25% of those suffering from ME are housebound or bedbound. The World Health Organization (WHO) classifies ME as a neurological disease.